Susanna C. Larsson

Susanna C. Larsson is affiliated with the Karolinska Institute.[1]She specializes in epidemiology.[2]She is a member of the Institute of Environmental Medicine.[3]

Larsson is an associate professor of epidemiology at the Karolinska Institute in Sweden.[4]

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the Karolinska Institute


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New study reveals that Coffee doesn't risk your heart

Lead author Susanna Larsson, from Karolinska Institute, Sweden, said that they found no evidence that high consumption of coffee increases the risk of atrial fibrillation. A new study has found no association between coffee consumption and an increased risk of atrial fibrillation. AF is the most frequent form of irregular heartbeat, causing a substantially increased risk of stroke, heart failure and all-cause mortality. It has previously been speculated that high coffee consumption may increase the risk of developing atrial fibrillation.[7]


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Increased HF Risk Tied to Soft Drinks (CME/CE)

High consumption of sweetened drinks was linked to increased risk for heart failure in middle-aged and older men, according to a Swedish study. Individuals reporting consumption of two or more sweetened beverages daily in the men-only study showed an adjusted hazard ratio for incident heart failure of 1.23 , relative to those reporting no such drinks in their diets, according to Susanna Larsson, PhD, of the Karolinska Institute in Stockholm, and colleagues, writing in the journal Heart. Previous studies have linked sweetened beverages with obesity, diabetes, hypertension, the metabolic syndrome, coronary heart disease, and stroke, Larsson and colleagues indicated. "High consumption of sweetened beverages may also increase the risk for heart failure," they said. "Our study findings suggest that sweetened beverage consumption could contribute to heart failure development," she and colleagues concluded.[6]


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Boozing may lead to some particular types of stroke

Susanna Larsson, lead author said, "This is the first study that combines the results from all available prospective studies on alcohol consumption and risk of hemorrhagic stroke subtypes. The association between heavy alcohol consumption and these two types of stroke was stronger than that for ischemic stroke." stroke is caused by blood clots which block diseased or damaged cerebral arteries. The researchers suggest that different associations between alcohol consumption and type of stroke may have to do with the effects alcohol has on the human body. research has found an association between alcohol consumption and lower levels of fibrinogen -- a protein in the body which helps the formation of blood clots. However, observational studies can show a possible association between alcohol consumption and risk of different types of stroke, but they cannot show cause and effect because other factors may have impacted the results. study has been published in BMC Medicine. story has not been edited by Business Standard staff and is auto-generated from a syndicated feed.)[5]


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Eating fried potatoes linked to higher risk of death, study says

Eating potatoes that have not been fried was not linked to a similar early mortality risk, the researchers noted. Susanna Larsson, an associate professor at the Institute of Environmental Medicine, Karolinska Institutet in Stockholm, Sweden, noted that the new study provides "no evidence" that potato consumption in and of itself may increase the risk of an early death. "Fried potato consumption may be an indicator of a less healthy dietary pattern which is associated with increased mortality," said she, who also conducted a study of potato consumption. Her study did not find an increased risk of cardiovascular disease linked to eating potatoes.[8]


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Nuts May Be Good for the Heart, but Are Hardly a Miracle Food

Nut consumption was associated with lowered risk for heart attack, heart failure, stroke and the irregular rapid heartbeat called atrial fibrillation, or A-fib. When the researchers controlled for these factors, nut consumption was associated only with a lower risk for A-fib, and had no significant effect on the other cardiovascular diseases. “It’s possible that previous studies didn’t control for as many factors as we did,” said the lead author, Susanna C. Larsson, an associate professor of epidemiology at the Karolinska Institute.[1]


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